Full Download Clitocybin B inhibits rat aortic smooth muscle cell proliferation through suppressing PDGF-Rb phosphorylation - K D Yoo; E S Park; Y Lim; S I Kang; S H Yoo; All authors file in ePub
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Clitocybin B inhibits rat aortic smooth muscle cell
Clitocybin B inhibits rat aortic smooth muscle cell proliferation through suppressing PDGF-Rb phosphorylation
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Carvacrol inhibits atherosclerotic neointima formation by
Tangeretin, a citrus flavonoid, inhibits PGDF-BB-induced
O-GlcNAc Modification of NFκB p65 Inhibits TNF-α-Induced
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Vinpocetine inhibits NF-κB dependent inflammation via an IKK
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Nitric oxide prevents aortic valve calcification by S
Astragaloside IV inhibits platelet‑derived growth factor‑BB
The Direct Effect of Levobupivacaine in Isolated Rat Aorta I
Increased risk of aortic aneurysm and dissection in patients
D-erythro-N,N-dimethylsphingosine inhibits bFGF-induced
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Human scalp follicles, mouse or rat vibrissae follicles, macaque and mink skin tissues. Modulation of mechanistic markers such as alp, wnt/b catenin pathway, jnk *clitocybin.
Quiescent rat aortic smcs were pretreated with glcn (5 mm), pugnac (10 −4 m) or vehicle and then stimulated with tnf-α (10 ng/ml). Both treatments inhibited tnf-α-induced expression of chemokines [cytokine-induced neutrophil chemoattractant (cinc)-2β and monocyte chemotactic protein (mcp)-1] and adhesion molecules [vascular cell adhesion molecule (vcam)-1.
2, pleurone, a novel human neutrophil elastase inhibitor f 이익수, 2011 5, clitocybin b inhibits rat aortic smooth muscle cell pro 유규동, 2012.
Dey nb, boerth nj, murphy- ullrich, chang pl, prince cw, lincoln tm (1998) cyclic gmp dependent protein kinase inhibits osteopontin and thrombospondin production in rat aortic smooth muscle cells.
Neutrophil elastase, leukocyte elastase, granulocyte elastase, pmn elastase, elane, pmn-elastase, polymorphonuclear leukocyte elastase, polymorphonuclear.
This study aims to investigate the effects of honokiol on proliferation, cell cycle, and apoptosis in tumor necrosis factor (tnf)-α-induced rat aortic smooth muscle cells (rasmcs). We found that honokiol treatment showed potent inhibitory effects on tnf-α-induced rasmc proliferation, which were associated with g0/g1 cell cycle arrest and downregulation of cell cycle-related proteins.
Blueberries are rich in anthocyanins (acns), which have recently been noted to protect against atherosclerosis development in mice. Cyanidin-3-o-glucoside (c3g), a member of blueberry acn family, can inhibit the tumor necrosis factor-α (tnf-α)-induced proliferation of vascular smooth muscle cells (vsmcs). However, the effects of c3g on vsmc apoptosis and migration remain unclear.
Inhibits rat aortic smooth muscle cell prolif eration through suppressing clitocybin b was reported to have either a potent free radical scavenging effect or effects that were isolated from.
Downregulation of poldip2 in rat aortic smooth muscle cells significantly reduced serum-induced proliferation and pcna expression, but upregulated p21 expression.
Zheng l, cao y, liu s, peng z and zhang s: neferine inhibits angiotensin ii‑induced rat aortic smooth muscle cell proliferation predominantly by downregulating fractalkine gene expression.
Feb 11, 2021 lithospermate b from salvia miltiorrhiza bunge: inhibition of mmps via ros scavenging and anti-wrinkle effects of clitocybin a isolated from the aortic smooth muscle cells from spontaneously hypertensive rats.
Inhibits the induction of nos activity in rat alveolar macrophages. Prevents apoptosis in human hl-60 cells and thymocytes, but induces apoptosis in human and rat smooth muscle cells.
Meso-dihydroguaiaretic acid inhibits rat aortic vascular smooth muscle cell proliferation by suppressing phosphorylation of platelet-derived growth factor receptor beta eur j pharmacol 2014 dec 5;744:36-41.
Rat vascular smooth muscle cells (vsmcs) were obtained from the thoracic aorta of male sprague-dawley rats (8 wk old) by the explantation method as described previously and cultured in growth medium (dulbecco's modified eagle's medium, invitrogen, carlsbad, ca) containing 10% fetal bovine serum at 37°c.
Tangeretin, a citrus flavonoid, inhibits pgdf-bb-induced proliferation and migration of aortic smooth muscle cells by blocking akt activation. Author information: (1)department of biochemistry, college of natural sciences, kangwon national university, chuncheon 200-701, republic of korea.
Anticancer bioactive foods inhibiting matrix metalloproteinases. Anticancer conclusion acknowledgments references further reading.
Meng z, yan c, deng q, gao df and niu xl: curcumin inhibits lps-induced inflammation in rat vascular smooth muscle cells in vitro via ros-relative tlr4-mapk/nf-κb pathways.
A critical role of nicotinamide phosphoribosyltranferase in human telomerase reverse transcriptase induction by resveratrol in aortic smooth muscle cells. Effects of chalcone derivatives and curcumin analogues on lipopolysaccharide-induced chemokines production.
Piceatannol has beneficial effects in many major diseases, including cancer and neurodegenerative diseases. 14, 17 piceatannol is well known as an inhibitor of spleen tyrosine kinase (syk) and inhibits the migration of rat aortic smooth muscle cells (rasmcs) by attenuating p38 mitogen-activated protein kinase (mapk)/hsp27 pathways.
We investigated the effects of carvacrol on the migration and proliferation of rat aortic smooth muscle cells (rasmcs) and on vascular neointima formation. Methods and results: carvacrol significantly inhibited platelet-derived growth factor (pdgf)-bb-stimulated rasmc migration and proliferation in a concentration-dependent manner.
The role of sphingosine kinase (sphk) on basic fibroblast growth factor (bfgf)-induced proliferation of cerebral, aortic and coronary smooth muscle cells (smc) was addressed using d-erythro-n,n-dimethylsphingosine (dms), an inhibitor of sphk which blocks conversion of sphingosine to sphingosine-1-phosphate (s1p).
Pterostilbene, a natural dimethylated analog of resveratrol, inhibits rat aortic vascular smooth muscle cell proliferation by blocking akt-dependent pathway.
Liensinine belongs to bisbenzylsioquinolines alkaloids derived from the lotus (nelumbo nucifera) possessing many pharmacological activities like anti-hypert ensive, anti-arrhythmic, anti-aging and anti-obesity. The key steps involve in the pathogenesis of atherosclerosis include free radical induce lipid peroxidation, inflammation due to interleukin-6 (il-6) and others inflammatory mediators.
However, the effect of clitocybin b on smooth muscle cell proliferation has not yet been clarified. The aim of this study was to elucidate the antiproliferative activity and the mechanistic target of clitocybin b on pdgf-bb-stimulated rat aortic vsmcs.
Genistein inhibits rat aortic smooth muscle cell proliferation through the induction of p27kip1. Spinal cord injury stimulated mitogenic signals to induce a serine-threonine kinase kis (kinase interacting stathmin) to phosphorylate p27kip1 on serine-10, so that p27kip1 could bind to crm1 and be exported from nuclei for degradation.
In patients with chronic kidney disease (ckd) or those undergoing hemodialysis, pathological calcific deposition known as ectopic calcification occurs in soft tissue, resulting in a life-threatening disorder. A potent and effective inhibitor of ectopic calcification is eagerly expected. In the current study, the effects of fyb-931, a novel bisphosphonate compound synthesized for the prevention.
Morphogenesis of vascular smooth muscle in atherosclerosis and cell culture.
Rat aortic mcp-1 and its receptor ccr2 increase with age and alter vascular smooth muscle cell function.
Calcific aortic valve disease (cavd) is an increasingly prevalent condition, and endothelial dysfunction is implicated in its etiology. We previously identified nitric oxide (no) as a calcification inhibitor by its activation of notch1 which is genetically linked to human cavd. Here, we show no rescues calcification by an s-nitrosylation–mediated mechanism in porcine aortic valve.
Nov 9, 2011 ㆍphage-mediated inhibition of bacterial group behaviors in pseudomonas ㆍ antiproliferative activity of clitocybin c, a novel isoindolinone, is mediated by blocking erk1/2 phosphorylation in rat aortic smooth muscle cell.
Yap1 up-regulation inhibits apoptosis of aortic dissection vascular smooth muscle cells the expression and significance of il-6, ifn-γ, sm22α, and mmp-2 in rat model of aortic dissection microrna-370-3p inhibits human vascular smooth muscle cell proliferation via targeting kdr/akt signaling pathway in cerebral aneurysm.
A role for p38 mitogen-activated protein kinase and c-myc in endothelin-dependent rat aortic smooth muscle cell proliferation. C-type natriuretic peptide down-regulates expression of its cognate receptor in rat aortic smooth muscle cells. 1,25-dihydroxyvitamin d inhibits human anp gene promoter activity.
Abdulhakim b, federica p, riccardo c, giuseppe v (2014) hypogeous fungi in mediterranean. Maquis protease inhibiting activities from fruiting bodies of cordyceps militaris.
Ekshyyan vp, hebert vy, khandelwal a and dugas tr: resveratrol inhibits rat aortic vascular smooth muscle cell proliferation via estrogen receptor dependent nitric oxide production.
Oct 22, 2020 our findings also indicate that these cdk inhibitors inhibit gsk3β, representing a potential reticulum stress and activation of gsk-3 in cultured rat retinal cells paeoniflorin blocks the proliferation of vascular.
Clitocybin b significantly inhibited the proliferation and the dna synthesis of pdgf-bb-stimulated vsmcs in a concentration-dependent manner. In agreement with these findings, clitocybin b suppressed the pdgf-bb-induced progression through g0/g1 to s phase of cell cycle.
Lidocaine is an approved local anesthetic and class 1b antiarrhythmic with a number of ancillary properties. Our aim was to investigate lidocaine’s vasoreactivity properties in intact versus denuded rat thoracic aortic rings, and the effect of inhibitors of nitric oxide (no), prostenoids, voltage-dependent kv and katp channels, membrane na+/k+ pump, and a2a and a2b receptors.
Clitocybin b inhibits rat aortic smooth muscle cell proliferation through suppressing pdgf-rβ phosphorylation has a variety of pharmacological effects, its effects on rat aortic smooth muscle.
A, b, the maximal wall area (a) and the maximal thickness (b) of the inner-aortic-arch wall for each mouse was measured as described in the methods.
17880 ensembl ensg00000133392 ensg00000276480 ensmusg00000018830 uniprot p35749 o08638 refseq (mrna) nm_001040113 nm_001040114 nm_002474 nm_022844 nm_001161775 nm_013607 refseq (protein) np_001035202 np_001035203 np_002465 np_074035 np_002465.
Objectivevascular calcification is a common pathobiological process which occurs among the elder population and in patients with diabetes and chronic kidney disease. Osteoprotegerin, a secreted glycoprotein that regulates bone mass, has recently emerged as an important regulator of the development of vascular calcification.
In the present study, we investigated whether compound c influences vascular smooth muscle cell (smc) function through the ampk pathway. 02–10 μm) inhibited vascular smc proliferation and migration in a concentration-dependent fashion.
This study is to determine the effect of the natural product parthenolide, a sesquiterpene lactone isolated from extracts of the herb tanacetum parthenium, on the proliferation of vascular smooth muscle cells (vsmcs). Rat aortic vsmcs were isolated and cultured in vitro, and treated with different concentrations of parthenolide (10, 20 and 30 μmol/l).
Park es, kang si, yoo kd, et al: camptothecin inhibits platelet-derived growth factor-bb-induced proliferation of rat aortic vascular smooth muscle cells through inhibition of pi3k/akt signaling pathway.
Clitocybin b inhibits rat aortic smooth muscle cell proliferation through suppressing pdgf-rβ phosphorylation.
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